Macerat 13 je biljni pripravak pelina, klinčića i crnog oraha potopljenih (maceriranih) u hladno prešanom ulju oraha.
Djelovanje ovog pripravka zasniva se na biljkama koje su šest tjedana uronjene u hladno prešano ulje oraha, čime na sebe veže njihova ljekovita svojstva.
Prema riječima fitoterapeuta Ive Bačlije:
"Pelin će zagorčati život parazitima, crni će ih orah zaviti u crno, a klinčić će im nemilosrdno uništiti čak i nerazvijena jajašca".
Macerat 13 - ubojit za parazite, a nježan za organizam.
Što kaže znanost
- Crni orah, odnosno ljuska crnog oraha sadrži prirodne kemikalije koje inhibiraju rast bakterija i gljivica (1). Crni orah sadrži impresivni profil hranjivih tvari a jedna od glavnih komponenti ljuske crnog oraha je juglon. Ljuska crnog oraha sadrži tanine, spojeve kojima su dokazana antibakterijska svojstva (2).
- Pelin se povijesno upotrebljava kao efikasno biljno sredstvo protiv parazita (3). Istraživanja pokazuju da pelin pomaže pri probavi i smanjuje grčeve u crijevima i želucu (4) čime se smanjuju gastrointestinalni simptomi poput proljeva, nadutosti, žgaravice, zatvora i plinova (5).
- Klinčići su bogati antioksidansima (6) pa mogu smanjiti nastanak kroničnih bolesti. Pokazalo se da imaju antimikrobna svojstva (7), odnosno pomažu u zaustavljanju rasta mikroorganizama.
- Ulje oraha je esencija najzdravijih orašastih plodova na svijetu. Studije potvrđuju da hladnim prešanjem ulje oraha zadržava tvari potrebne za zdravlje. Redovita i adekvatna konzumacija oraha povezana je sa smanjenjem rizik od bolesti kao što su karcinomi (8,9), kardiovaskularnih bolesti (10,11,12,13,14,15,16,17), dijabetesa (18) i degenerativnih poremećaja (19,20).
Reference na znanstvene radove o učinku navedenih biljaka pronađite u tabu "Reference".
Reference
1) Ho KV, Lei Z, Sumner LW, et al. Identifying Antibacterial Compounds in Black Walnuts (Juglans nigra) Using a Metabolomics Approach. Metabolites. 2018;8(4):58. Published 2018 Sep 29. doi:10.3390/metabo8040058.
2) Serrano J, Puupponen-Pimiä R, Dauer A, Aura AM, Saura-Calixto F. Tannins: current knowledge of food sources, intake, bioavailability and biological effects. Mol Nutr Food Res. 2009 Sep;53 Suppl 2:S310-29. doi: 10.1002/mnfr.200900039. PMID: 19437486.
3) Therapeutic efficacy of Artemisia absinthium against Hymenolepis nana: in vitro and in vivo studies in comparison with the anthelmintic praziquantel. J Helminthol. 2018;92(3):298-308. doi:10.1017/s0022149x17000529
4) Almario CV, Ballal ML, Chey WD, Nordstrom C, Khanna D, Spiegel BMR. Burden of gastrointestinal symptoms in the United States: Results of a nationally representative survey of over 71,000 Americans. Am J Gastroenterol. 2018;113(11):1701-1710. doi:10.1038/s41395-018-0256-8
5) Mcmullen MK, Whitehouse JM, Whitton PA, Towell A. Bitter tastants alter gastric-phase postprandial haemodynamics. J Ethnopharmacol. 2014;154(3):719-27. doi:10.1016/j.jep.2014.04.041
6) Shan B, Cai YZ, Sun M, Corke H. Antioxidant capacity of 26 spice extracts and characterization of their phenolic constituents. J Agric Food Chem. 2005 Oct 5;53(20):7749-59. doi: 10.1021/jf051513y. PMID: 16190627.
7) Nzeako BC, Al-Kharousi ZS, Al-Mahrooqui Z. Antimicrobial activities of clove and thyme extracts. Sultan Qaboos Univ Med J. 2006;6(1):33-39.
8) Yang J., Liu R.H., Halim L. Antioxidant and antiproliferative activities of common edible nut seeds. LWT-Food Sci. Technol. 2009;42:1– doi: 10.1016/j.lwt.2008.07.007.
9) Nagel J.M., Brinkoetter M., Magkos F., Liu X., Chamberland J.P., Shah S., Zhou J., Blackburn G., Mantzoros C.S. Dietary walnuts inhibit colorectal cancer growth in mice by suppressing angiogenesis. Nutrition. 2012;28:67–75. doi: 10.1016/j.nut.2011.03.004.
10) Anderson K.J., Teuber S.S., Gobeille A., Cremin P., Waterhouse A.L., Steinberg F.M. Walnut polyphenolics inhibit in vitro human plasma and LDL oxidation. J. Nutr. 2001;131:2837–2842. doi: 1093/jn/131.11.2837.
11) Feldman E.B. The scientific evidence for a beneficial health relationship between walnuts and coronary heart disease. J. Nutr. 2002;132:1062S–1101S. doi: 10.1093/jn/132.5.1062S
12) Fraser G.E., Sabate J., Beeson W.L., Strahan T.M. A possible protective effect of nut consumption on risk of coronary heart disease: The Adventist Health Study. Arch. Intern. Med. 1992;152:1416. doi: 10.1001/archinte.1992.00400190054010
13) Holt R., Yim S.J., Shearer G., Keen C., Djurica D., Newman J., Shindel A., Hackman R. Correlation of lipoprotein epoxide content to microvascular function after short-term walnut intake (831.5) FASEB J. 2014;28:831–835.
14) Horton K., Morgan J., Uhrin L., Boyle M., Altomare P., Laskowsky C., Walker K., Stanton M., Newman L., Capuzzi D. The effect of walnuts on serum lipids consumed as part of the national cholesterol educational panel step 1 diet. J. Am. Diet. Assoc. 1999;99:A109. doi: 10.1016/S0002-8223(99)00786-5.
15) Hu F.B., Stampfer M.J. Nut consumption and risk of coronary heart disease: A review of epidemiologic evidence. Curr. Atheroscler. Rep. 1999;1:204–209. doi: 10.1007/s11883-999-0033-7.
16) Kris-Etherton P.M., Hu F.B., Ros E., Sabaté J. The role of tree nuts and peanuts in the prevention of coronary heart disease: Multiple potential mechanisms. J. Nutr. 2008;138:1746–1751. doi: 10.1093/jn/138.9.1746S.
17) Almario R.U., Vonghavaravat V., Wong R., Kasim-Karakas S.E. Effects of walnut consumption on plasma fatty acids and lipoproteins in combined hyperlipidemia. Am. J. Clin. Nutr. 2001;74:72–79. doi: 10.1093/ajcn/74.1.72.
18) Tapsell L.C., Gillen L.J., Patch C.S., Batterham M., Owen A., Baré M., Kennedy M. Including walnuts in a low-fat/modified-fat diet improves HDL cholesterol-to-total cholesterol ratios in patients with type 2 diabetes. Diabetes Care. 2004;27:2777–2783. doi: 10.2337/diacare.27.12.2777.
19) Chauhan A., Essa M.M., Muthaiyah B., Chauhan V., Kaur K., Lee M. Walnuts-rich diet improves memory deficits and learning skills in transgenic mouse model of Alzheimer’s disease. Alzheimers Dementia. 2010;6:S69. doi: 10.1016/j.jalz.2010.05.206.
20) Gorji N., Moeini R., Memariani Z. Almond, hazelnut and walnut, three nuts for neuroprotection in Alzheimer’s disease: A neuropharmacological review of their bioactive constituents. Pharmacol. Res. 2018;129:115–127. doi: 10.1016/j.phrs.2017.12.003
